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. 2022 Mar 9;18(11):2711–2730. doi: 10.1080/15548627.2022.2047344

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Figure 8. — Treatment with BafA1 reduced angiogenesis in the ipsilateral thalamus after cortical infarction. (A) Immunoblotting reveals MAP1LC3B-II conversion and SQSTM1 accumulation in vessels of the ipsilateral thalamus in the vehicle and BafA1 groups at 7 days after MCAO. (B and C) Quantitative analysis of MAP1LC3B-II or SQSTM1 expression relative to GAPDH. n = 4, data are expressed as median ± interquartile range. *P < 0.05, compared with the vehicle group. (D) Co-staining of BrdU (green) with LAMA1 (red) in the ipsilateral thalamus in the vehicle and BafA1 groups at 7 days after MCAO (arrows). Scale bar: 50 μm, 100 μm. (E and F) Quantitative analysis of BrdU⁺_LAMA1⁺ cells and vessel density. n = 6, data are expressed as median ± interquartile range. **P < 0.01, compared with the vehicle group.