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Proposed working model by which BCL6 is upregulated in pre-eclamptic placentas. Upregulation and activation of BCL6 in pre-eclamptic placentas may be caused at transcriptional, translational and post-translation level. HIF1, hypoxia-inducible factor 1; HSF1, heat shock factor 1; HSP90, heat shock protein 90; IL8, interleukin 8; lncRNA, long non-coding RNA; miRNA, microRNA; STAT, signal transducer and activator of transcription; TNFα, tumor necrosis factor alpha.
