Figure 5.
Increased BCL6 in endometriosis. Schematic illustration shows that various factors may upregulate and stabilize BCL6, which may impact vital cellular activities in diverse cell types of endometrial tissues through multiple signaling pathways, affecting the pathogenesis of endometriosis and its related infertility. ECM, extracellular matrix; FAK, focal adhesion kinase; GC, germinal center; IL6, interleukin 6; MAPK, mitogen-activated protein kinase; PI3K, phosphatidylinositol 3-kinase; STAT3, signal transducer and activator of transcription 3; TFH cells, T follicular helper cells.