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. 2021 Apr 14;224(10):1796–1805. doi: 10.1093/infdis/jiab207

Table 3.

Association Between Zygosity at HLA Class I Loci and Class II Loci and HCC Among REVEAL-HBV Cohort in Taiwan

HLA Zygosity Incident HCC Cases (n = 306) Chronic HBV Carriers Without HCC (n = 3110) HR Adjusted for Age, Sex, and Principal Components (95% CI)a HR Further Adjusted for Viral Load (95% CI)b
Class I loci
 Heterozygous at all loci 191 (62.4) 2118 (68.1) Ref. Ref.
 Homozygous in at least 1 locus 115 (37.6) 992 (31.9) 1.31 (1.04–1.65) 1.27 (1.00–1.60)
 Number of homozygous loci
  1 88 (28.8) 698 (22.4) 1.42 (1.10–1.83) 1.31 (1.02–1.68)
  2 18 (5.9) 200 (6.4) 0.99 (.61–1.60) 1.11 (.68–1.80)
  3 9 (2.9) 94 (3.0) 1.18 (.60–2.31) 1.25 (.64–2.45)
P trend .168 .130
 HLA-A
  Heterozygote 244 (79.7) 2533 (81.4) Ref. Ref.
  Homozygote 62 (20.3) 577 (18.6) 1.13 (.85–1.49) 1.13 (.86–1.50)
 HLA-B
  Heterozygote 271 (88.6) 2788 (89.6) Ref. Ref.
  Homozygote 35 (11.4) 322 (10.4) 1.11 (.78–1.58) 1.11 (.78–1.58)
 HLA-C
  Heterozygote 252 (82.4) 2629 (84.5) Ref. Ref.
  Homozygote 54 (17.6) 481 (15.5) 1.24 (.93–1.67) 1.27 (.95–1.71)
Class II loci
 Heterozygous at all loci 157 (51.3) 1708 (54.9) Ref.
 Homozygous in at least 1 locus 149 (48.7) 1402 (45.1) 1.21 (.97–1.51) 1.17 (.93–1.46)
  Number of homozygous
   1 101 (33.0) 1001 (32.2) 1.15 (.89–1.47) 1.13 (.88–1.46)
   2 27 (8.8) 241 (7.7) 1.21 (.80–1.81) 1.04 (.69–1.56)
   3 21 (6.9) 160 (5.1) 1.64 (1.04–2.59) 1.70 (1.08–2.70)
  P trend .031 .068
 HLA-DPB1
  Heterozygote 193 (63.1) 2010 (64.6) Ref. Ref.
  Homozygote 113 (36.9) 1100 (35.4) 1.16 (.92–1.46) 1.20 (.95–1.51)
 HLA-DQB1
  Heterozygote 240 (78.4) 2595 (83.4) Ref. Ref.
  Homozygote 66 (21.6) 515 (16.6) 1.40 (1.06–1.84) 1.21 (.92–1.60)
 HLA-DRB1
  Heterozygote 367 (87.3) 2762 (88.8) Ref. Ref.
  Homozygote 39 (12.7) 348 (11.2) 1.17 (.84–1.64) 1.15 (.82–1.61)

Data are No. (%).

Abbreviations: CI, confidence interval; HBV, hepatitis B virus; HCC, hepatocellular carcinoma; HR, hazard ratio; REVEAL-HBV, Risk Evaluation of Viral Load Elevation and Associated Liver Disease/Cancer-Hepatitis B Virus study.

aHRs and 95% CIs were estimated from Cox proportional hazards regression models with attained age as the time scale and were adjusted for sex and principal components. Censored at death, diagnosis of HCC, and end of follow-up (2016), whichever occurred first.

bAdditionally adjusted for baseline HBV DNA load (in categories, <300, 300–9999, 10 000–99 999, 100 000–999 999, ≥1 000 000 copies/mL). Subjects with missing on baseline HBV DNA load were retained in the analysis (n = 176).