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. 2022 Nov 4;8:444. doi: 10.1038/s41420-022-01238-4

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — A Landscape for transcript expression of GPR35 in pan-cancer. B The correlation between signature gene expression (angiogenesis, cell proliferation, and apoptosis) and GPR35 expression in GC. C Correlation coefficient between tumor stemness and GPR35 for different cancers. D GPR35 expression level in different GC cells. E Cell proliferation ability decreased after infected si-GPR35 (24 h). F si-GPR35 promotes cell apoptosis for MKN-27. The representative images and quantitative data of Transwell assays (G) and wound healing assays (H) of MKN-27 cells after different treatments, respectively.