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. 2022 Nov 4;8:444. doi: 10.1038/s41420-022-01238-4

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — A The representative images of M0 macrophages morphological change (from oval cells to antennal, spindle-shaped adherent cells) for different treated groups, i.e., si-GPR35, si-CTCF-bd of CAPN10, and negative control. B Quantitative data of M2 markers in different treated groups. C Relative positive rate for CD206 in different treated groups. D–H GPR35 levels in different cluster macrophages in gastric premalignant lesions and early gastric cancer. D Average expression for biomarkers of macrophage in clusters for cells of various gastric disease tissues. E–F UMAP (E) and bubble (F) diagram for GPR35 expression level in macrophage-01/02. G Sample composition for scRNA-seq. H GPR35 expression level for CTSB+ and CD68 + macrophage in various samples.