(A, B) Representative confocal images of cryoprotected adult dorsal root ganglia (DRG) sections (25 μm) with quantifications indicating the percentage of NaV+ and NaV- neurons in TdTomato+ proprioceptors. Sections were hybridized using RNAscope with probes targeting NaV1.6 or NaV1.7 (Scn8a and Scn9a, respectively, magenta) and stained with anti-DsRed (yellow). (A) Snc8a, n = 298, (B) Scn9a, n = 166. Scale bar set to 50 μm. White arrowheads indicate NaV+ neurons. (C–, D) Frequency distribution plots of integrated fluorescence density of NaV1.6 and NaV1.7 mRNA in TdTomato+ proprioceptors, respectively. (E) The average integrated density of Scn1a, Scn8a, and Scn9a RNAscope probe signal. Dots represent individual cells. Statistical significance was determined using a Kruskal–Wallis test with Dunn’s post-hoc comparisons. (F) Top: experimental workflow of serial pharmacological blockade of NaV channels expressed in proprioceptors. We first elicited a whole-cell sodium current in the absence of drug. We next bath applied 500 nM of ICA 121431 to block current carried by NaV1.1. Subsequently, we bath applied 9-anhydroustetrodoxin (AH-TTX) (300 nM) to block NaV1.6-mediated current, and PF-05089771 (25 nM) to block the NaV1.7-mediated current. Finally, tetrodotoxin (TTX) (300 nM) was used to block residual current and to confirm there was no contribution of TTX-resistant NaVs in proprioceptors. Bottom: representative current traces following application of NaV-selective inhibitors. All drugs were applied for 1 min. (G) Quantification of the average percentage of the whole-cell sodium current that was sensitive to the individual drugs used (n = 8). n = cells. ****p<0.0001.