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. 2022 Nov 8;13(11):941. doi: 10.1038/s41419-022-05391-7

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — a Local secondary structure of FMR1 mRNA at the regions targeted by the three FMR1 small interfering RNAs. b Western blot was used to validate the expression of FMR1 in CT26 cells with FMR1 shRNA. c Schematic of subcutaneous tumor formation assay in CT26 mice formed by CRC cells with FMR1 shRNA carried by attenuated Salmonella. d Images of the representative tumors in each group on posttreatment 14 days. e Tumor size analysis. f Tumor weight analysis. g Body weight analysis. h Proposed model underlying the effect of FMR1-medicated EGFR mRNA stability in colorectal cancer. Data are shown as means ± SD. *P < 0.05, **P < 0.01, ***P < 0.001.