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. 2022 Oct 28;14(21):4549. doi: 10.3390/nu14214549

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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L-cit attenuates oxidative stress induced by iron overload in mouse thymus. Mice were intraperitoneally injected with iron dextran 50 mg/kg, and L-cit was given by gavage administration with a dosage of 0.5, 1.0, and 2.0 g/kg/day. (A,B) CD8⁺ T-lymphocytes determined by immunofluorescence assay. (C) MDA concentration. (D) GSH-Px activity. (E) SOD activity. Values represent mean ± SEM. of three independent experiments. Significant differences are represented by * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001, ns (not significant).

L-cit attenuates oxidative stress induced by iron overload in mouse thymus. Mice were intraperitoneally injected with iron dextran 50 mg/kg, and L-cit was given by gavage administration with a dosage of 0.5, 1.0, and 2.0 g/kg/day. (A,B) CD8⁺ T-lymphocytes determined by immunofluorescence assay. (C) MDA concentration. (D) GSH-Px activity. (E) SOD activity. Values represent mean ± SEM. of three independent experiments. Significant differences are represented by * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001, ns (not significant).