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The interplay of oxidative stress and inflammation in MIS-C. SARS-CoV-2 induces pathological responses including increased inflammation, oxidative stress, and endothelial dysfunction, which contribute to progression into severe disease. TLR: Toll-like receptor; ACEr: angiotensin-converting enzyme receptor; ROS: reactive oxygen species; Nrf2: nuclear factor erythroid 2-related factor 2; NOXs: NADPH oxidase enzymes; IL: interleukin; Ig: immunoglobulin.
