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. 2022 Jul 21;73(20):7055–7067. doi: 10.1093/jxb/erac315

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© The Author(s) 2022. Published by Oxford University Press on behalf of the Society for Experimental Biology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 3. — Model for the SnRK1/TOR/FLZ signaling network. Under unfavorable conditions, both FLZ6 and FLZ10 repress SnRK1, allowing TOR signaling, in an ER localization manner. Under favorable conditions, FLZ8 induces TOR signaling inhibition by two different mechanisms, namely (i) promoting SnRK1 signaling through enhancing the SnRK1α1 level and (ii) stimulating RAPTOR1B–SnRK1α1 interaction in the ER. Lines with arrows indicate positive regulation and lines with bars indicate negative regulation. The figure was created with BioRender.