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. 2022 Nov 16;20:484. doi: 10.1186/s12951-022-01692-3

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Anti-tumor efficacy of BMT@LA. A (a) Chemical formula of key components in BMT@LA when performing SDT/gas treatment. b Schematic diagram of BMT@LA inhibition of primary and distant tumors in immunotherapy. B, C Primary and distant tumor volumes in tumor-bearing mice after receiving different treatment regimens. D Flow cytometry analysis of the proportion of mature DCs (CD11⁺CD80⁺CD86⁺ as markers), CD8⁺T cells (CD3⁺CD8⁺ as markers), and CD4⁺T cells (CD3⁺CD4⁺ as markers) in the spleen after different treatment regimens (Treatment groups: 1: Ctl, 2: US, 3: α-PD-L1, 4: BMT@LA, 5: LA + US, 6: BMT + US, 7: BMT@LA + US, 8: BMT@LA + US + α-PD-L1) Reprinted with permission from Ref. [176].

Copyright© 2021, copyright Wang et al.