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. 2022 Nov 3;16:1013712. doi: 10.3389/fnins.2022.1013712

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Copyright © 2022 Arai, Kanazawa, Kimura, Munakata, Yamakawa, Shinmura, Yuasa, Sano and Fukuda.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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New animal model of epilepsy-induced stress cardiomyopathy (SC). (A) Protocol for the induction of SC in Wistar rats using pilocarpine. (B) Electrocardiograms revealing typical ST elevation during SC. Pre, pre-inducing epilepsy; Post, post-inducing epilepsy; (C,F) Echocardiography (c) and left ventriculography (F) revealed that wall motion of the left ventricle (LV) apical segment was akinetic or dyskinetic during SC. (D) Incidence of epilepsy-induced SC (n = 9). (E) Upregulated serum epinephrine, norepinephrine, and dopamine levels 2 h after the induction of epilepsy (n = 4). Where appropriate, data are provided as the mean ± SD. *P < 0.05 compared with 0 min (E).