Figure 2.

A 72-year-old male patient with chronic hepatitis C virus (HCV) infection, liver cirrhosis, and high a-fetoprotein. Full standard protocol: A, B) axial DWI and ADC showed right lobe infiltrative hepatic focal lesion with true diffusion restriction. C, D) Axial T1 WI image in and out of phase: the lesion elicited a low signal. E) Axial T2WI image: the lesion elicited a high signal. Dynamic contrast enhancement (F, G, H): arterial phase image (F) portovenous phase (G), and delayed phase (H) revealed heterogenous arterial enhancement with delayed washout of the lesion with intravenous tumoural thrombus. The lesion was categorized as LI-RADS-TIV. Abbreviated 1: E) Axial T2WI image: the lesion elicited a high signal. Dynamic contrast enhancement (F, G, H): arterial phase image (F), portovenous phase (G), and delayed phase (H) revealed heterogenous arterial enhancement with delayed washout of the lesion with intravenous tumoral thrombus. The lesion was categorized as LI-RADS-TIV. Abbreviated 2: A, B) Axial DWI and ADC showed right lobe infiltrative hepatic focal lesion with true diffusion restriction. Dynamic contrast enhancement (F, G, H): arterial phase image (F), portovenous phase (G), and delayed phase (H) revealed heterogenous arterial enhancement with delayed washout of the lesion with intravenous tumoral thrombus. The lesion was categorized as LI-RADS-TIV. Abbreviated 3: (C, D) Axial T1 WI image in and out of phase: the lesion elicited a low signal. Dynamic contrast enhancement (F, G, H): arterial phase image (F) portovenous phase (G), and delayed phase (H) revealed heterogenous arterial enhancement with delayed washout of the lesion with intravenous tumoural thrombus. The lesion was categorized as LI-RADS-TIV