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. 2022 Nov 19;20:159. doi: 10.1186/s12958-022-01036-8

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — CXCL3 as a downstream effector of TARID. a Scatter plot comparing expression levels between groups. b Differential gene cluster map. c KEGG metabolic pathway map of up-regulated genes. d, e The transcription level of TARID target genes in HTR8/SVneo cells with TARID overexpression (d) and knockdown (e). f Western blotting results for CXCL3 in HTR8/SVneo cells with TARID overexpression and knockdown. g, h Results of real-time quantitative PCR (g) and western blotting (h) showing the expression of CXCL3 in HTR8/Svneo cells transiently transfected with vector-CXCL3 or vector and si-CXCL3 or si-NC. i Results of real-time quantitative PCR showing the expression of TARID in HTR8/Svneo cells transiently transfected with vector-CXCL3 or vector and si-CXCL3 or si-NC. All experiments were conducted in triplicate; the values are shown as the mean ± SEM; vs. vector or ASO，**P < 0.01, *P < 0.05