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. 2022 Nov 7;13:1032361. doi: 10.3389/fendo.2022.1032361

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Copyright © 2022 Arefhosseini, Ebrahimi-Mameghani, Najafipour and Tutunchi

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Established pathophysiological mechanisms linking PCOS and NAFLD. AMPK, Adenosine monophosphate-activated protein kinase; DHT, Dihydrotestosterone; FFA, Free fatty acid; FSH, follicle-stimulating hormone; HA, Hyperandrogenism; IR, Insulin resistance; LDLR, Low-density lipoprotein receptor; LH, Luteinizing hormone; NAFLD, Nonalcoholic fatty liver disease; NASH, Non-alcoholic steatohepatitis; OS, Oxidative stress; PCOS, Polycystic ovarian syndrome; PPARα, Peroxisome proliferator-activated receptor-α; SHBG, Sex hormone-binding globulin.