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. 2022 Nov 7;13:1053330. doi: 10.3389/fmicb.2022.1053330

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Copyright © 2022 Zhou, Yong, Zhu, Yang and Fang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effect of exogenous D-ribose on the electron transport chain. (A) The intracellular NADH concentration of SCH-R and clinical isolates by D-ribose dose. (B) Activity of NADH dehydrogenase 1 in SCH-R and clinical isolates by D-ribose dose. (C) Percent survival of SCH-R and clinical isolates by rotenone dose and 1 MIC gentamicin plus 24 mM D-ribose. (D) QRT-PCR of key electron transport chain genes in the presence 24 mM D-ribose. nuoI, NADH dehydrogenase I chain I; ndh, NADH:quinone reductase; cyoB, cytochrome o ubiquinol oxidase subunit I; cydA, cytochrome BD2 subunit I; atpA, atpD, atpG, atpH and atpC, membrane-bound ATP synthase, F1 sector, alpha-subunit, beta-subunit, gamma-subunit, delta-subunit, and epsilon-subunit. Results are displayed as the mean ± SEM and three biological repeats are carried out. Significant differences are identified (*p < 0.05 and **p < 0.01).