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. 2022 Nov 21;24:81. doi: 10.1186/s13058-022-01580-6

Table 2.

Multivariate analysis of clinicopathological model, Model 2 (the early-treatment model) and Model 3 (the posttreatment model)

Clinicopathological model p-value Model 2 p-value Model 3 p-value
OR 95%CI OR 95%CI OR 95%CI
Ki67
  Negative Reference
  Positive 1.436 0.783–2.633 0.240 1.716 0.815–3.615 0.153 1.985 0.858–4.594 0.107
ER
  Negative Reference
  Positive 0.742 0.333–1.657 0.465 0.559 0.212–1.470 0.236 0.700 0.242–2.024 0.509
PR
  Negative Reference
  Positive 0.990 0.427–2.302 0.984 0.701 0.258–1.904 0.484 0.768 0.231–2.548 0.664
Her2
  Negative Reference
  Positive 0.937 0.078–11.320 0.959 1.062 0.369–3.055 0.911 1.036 0.063–16.92 0.980
Subtype
  Triple-negative Reference
  HER2(+) 2.047 0.161–25.97 0.578 1.375 0.083–22.90 0.823 1.271 0.075–21.44 0.867
  HER2(−)&HR(+) 1.194 0.361–3.945 0.770 1.275 0.305–5.331 0.738 1.378 0.272–6.987 0.697
P0-Signature 2.357 1.531–3.629  < 0.001 1.881 1.222–2.894 0.004
P1-Signature 2.455 1.721–3.502  < 0.001 2.094 1.468–2.987  < 0.001
P2-Signature 2.097 1.543–2.849  < 0.001

Model 2 was built based on the P0-Signature plus the P1-Signature. And Model 3 was based on Model 2 plus the P2-Signature

ER estrogen receptor, PR progesterone receptor, HER2 human epidermal growth factor receptor, P0 Phase 0 (pretreatment), P1 Phase 1 (early-stage treatment, namely, during the first–second cycle of the neoadjuvant chemotherapy), P2 Phase 2 (posttreatment), OR odds ratio, 95%CI 95% confidence interval