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. 2022 Nov 9;12:1028267. doi: 10.3389/fcimb.2022.1028267

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Copyright © 2022 Wu, Xu, Wang, Chen, Lv, Zheng, Yan, Jiang, Shen, Wang, Wang, Xia, Han and Li

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Pectin protects mice with CDI from intestinal infection by enhancing AhR pathway activation. (A, B) Relative mRNA levels of IL-22 and AhR in colon tissue. *, P<0.05; **, P<0.01; ***, P<0.001; ns, differences are not significant. (C, D) Body weight changes and survival curves of the mice with CDI treated with the AhR antagonist CH223191. *, CCDI vs PCDI+ CH223191, P<0.05; ^#, PCDI vs PCDI+ CH223191, P<0.05. (E) Relative mRNA levels of ZO-1 in colon tissues. (F, G) Body weight changes and survival curves of the mice with CDI treated with the AhR agonist FICZ. ^#, CCDI vs CCDI+FIZC, P<0.05. (H) Relative mRNA levels of ZO-1 in colon tissues.