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. 2021 Jul 9;20(2):260–273. doi: 10.1016/j.gpb.2021.07.001

Figure 4.

Figure 4

Comparison of non-synonymous SNPs in ARGs and VirGs between periodontitis and control samples from both datasets

A. Distribution of the MAF of non-synonymous SNPs in ARGs from both the current study and a study by Wang and colleagues [24]. Comparison of the MAF of non-synonymous SNPs in ARGs between periodontitis and control samples in both datasets. B. Distribution of the MAF of unique non-synonymous SNPs in ARGs from both the current study and a study by Wang and colleagues [24]. Comparison of the MAF of unique non-synonymous SNPs in ARGs between periodontitis and control samples in both datasets. C. Distribution of the MAF of non-synonymous SNPs in VirGs from both the current study and a study by Wang and colleagues [24]. Comparison of the MAF of non-synonymous SNPs in VirGs between periodontitis and control samples in both datasets. D. Distribution of the MAF of unique non-synonymous SNPs in VirGs from both the current study and a study by Wang and colleagues [24]. Comparison of the MAF of unique non-synonymous SNPs in VirGs between periodontitis and control samples in both datasets. All P values were calculated using Wilcoxon rank sum test. SNP, single-nucleotide polymorphism; ARG, antibiotic resistance gene; VirG, virulence gene; MAF, minor allele frequency.