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. 2022 Mar 3;24(4):415–423. doi: 10.1007/s12017-022-08705-2

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Upregulation of proinflammatory markers and co-stimulatory factors in GF23-2D2 mice A The transcriptome analysis showed the mRNA levels of several proinflammatory markers and co-stimulatory factors of the cerebellum of GF23-2D2, GF-IL23, 2D2, and WT mice. B The mRNA levels of targets involved in Th17 and Th1 cell signaling are listed from GF23-2D2 mice in relation to 2D2 and GF-IL23 with *p < 0.05. CD cluster of differentiation, CCL chemokine (C–C motif) ligand, CCR C–C chemokine receptor type, CXCL chemokine (C-X-C motif) ligand, H2-Eb1 histocompatibility 2, class II antigen E beta, Icam1 intercellular adhesion molecule 1, IFNγ interferon gamma, IL Interleukin