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. 2022 Nov 10;16:817732. doi: 10.3389/fncel.2022.817732

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Copyright © 2022 Qin, Zhang, Zhao, Liu, Qiu, Gong, Fan, Guo, Guo, Xu and Guo.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Schematic representation showing the modulation of neuroplasticity by acupuncture after ischemic stroke by the promotion of functional compensation and structural plasticity. The data of acupoint selection and mechanism are derived from the mechanistic studies by using human and rodents. PC6, Neiguan; GV26, Shuigou; GV24, Shenting; GV20, Baihui; GB7, Qubin; GB20, Fengchi; GV14, Dazhui; LI11, Quchi; LI10, Shousanli; TE5, Waiguan; LI4, Hegu; GB33, Xiyangguan; GB34, Yanglingquan; ST36, Zusanli; ST37, Shangjuxu; GB39, Xuanzhong; LR3, Taichong; KI3, Taixi; SP6, Sanyinjiao; SP10, Xuehai; EX-UE9, Baxie; NSCs, neural stem cells; NGF, nerve growth factor; BDNF, brain-derived neurotrophic factor; GAP-43, growth associated protein-43; SYN, synaptophysin; NR1, N-methyl-d-aspartate receptor subtype 1; TRPV1, transient receptor potential vanilloid subtype 1; PSD-95, postsynaptic density-95; LTP, long-term potentiation.