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. 2022 Nov 23;15:123. doi: 10.1186/s13048-022-01059-0

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Analysis of the TME, immune cell infiltration and immune checkpoints. A The proportion of 22 immune cells built on 3 clusters; B The difference of the fraction of 22 immune cells in 3 clusters; C The relationship between the proportion of dendritic cells resting and survival; D The relationship between the proportion of macrophages M2 cells and survival; E-G The relationship between the expression level of PDCD1, CD274, CTLA4 and survival; H The expression of 6 immune checkpoint genes with significant differences among the 3 clusters; I-L The ESTIMATE Score, Immune Score, Stromal Score, Tumor Purity were significantly different between 3 clusters. (*p < 0.05, **p < 0.01, ***p < 0.001, ns: nonsignificance)