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. 2022 Nov 24;22:308. doi: 10.1186/s12906-022-03794-9

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — WEA improves hepatic lipid metabolic gene expression in NAFLD rats. The liver samples were collected from the rats in Fig. 2. The hepatic mRNA expression of SREBP1c (A), SCD1 (B), FASN (C), and PPARα (D) were tested using real-time PCR assay. The data are expressed as mean ± SD, n = 8 per group. ^**P < 0.01 vs. Control group; ^#P < 0.05, ^##P < 0.01 vs. HFD group; ^$P < 0.04 vs. HFD + WEA 0.4 group. SREBP1c: sterol regulatory element binding protein 1c; SCD1: stearoyl-coA desaturase 1; FASN: fatty acid synthase; PPARα: peroxisome proliferator-activated receptor alpha