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. 2022 Nov 6;11(11):2195. doi: 10.3390/antiox11112195

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Liver mitochondrial biogenesis and β-oxidation related proteins in the four groups of rats studied: Sham, Sham + curcumin (ShamC), five-sixths nephrectomy (5/6Nx), and 5/6Nx + curcumin (5/6NxC). (A) Peroxisome proliferator-activated receptor γ co-activator 1 α (PGC1α), (B) Peroxisome proliferator-activated receptor α (PPARα), (C) voltage-dependent anion channel (VDAC), (D) Palmitoyl carnitine transferase 1 (CPT1), (E) ATP synthase 5a (ATP5a) and (F) representative images of western blot. β-actin was used as a loading control One-way analysis of variance (ANOVA), post hoc Fisher´s Least significant difference (LSD), Mean ± SD, n = 3. ^$ p < 0.05 vs. Sham, ^& p < 0.05 vs. ShamC, ^# p < 0.05 vs. 5/6Nx.