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. 2022 Nov 12;15(11):1394. doi: 10.3390/ph15111394

Figure 5.

Figure 5

Expression of inflammation-related proteins after treatment. (A) Western blotting analysis of collagen I, p38, p-p38, and Smad3 expression levels in lung tissues after different treatments (n = 3). (B–D) Analysis of the expression of collagen I, p-p38/p38, and Smad3 in the lungs after different treatments (n = 3). Quantification was performed using Image J software. * p < 0.05, ** p < 0.01. (E) HYAP improved BLM-induced lung fibrosis by blocking the MAPK-p38 and TGF-β/Smad signaling pathways, thereby inhibiting secretion of related inflammatory factors and fibroblasts’ proliferation.