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. 2022 Nov 25;5:1292. doi: 10.1038/s42003-022-04241-8

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Spearman correlation of H4K20me1 and H4K20me3 regions in PARP1 knockdown HeLa cells and its control. b Genome-wide metagene plot showing H4K20me1 profile (ChIP-seq) in control and PARP1 knockdown HeLa cells. c Genome-wide metagene plot showing H4K20me3 profile (ChIP-seq) in control and PARP1 knockdown HeLa cells. d Peak width profile of H4K20me1 by binning the peaks into different lengths in PARP1 knockdown cells and its control. e Peak width profile of H4K20me3 by binning the peaks into different lengths in PARP1 knockdown cells and its control. f Representative IGV genomic tracks showing H4K20me1 and RNAseq profile in PARP1 knockdown cells and its control.