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. 2022 Aug 22;48(1):54–60. doi: 10.1038/s41386-022-01411-4

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Figure depicts the impact of synaptic transmission blockade on the rapidity of homeostatic plasticity. While classical experiments relying on activity blockade (via blockade of voltage-gated Na⁺ channels or postsynaptic AMPA receptors) demonstrated a slow process that occurs over days, block of synaptic NMDA receptor facilitates plasticity down to hours or less.