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. 2022 Apr 15;15(6):1257–1269. doi: 10.1038/s41385-022-00513-y

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Il10gfp-foxp3rfp B6 mice were infected with 200 L3 H. polygyrus and 7 days later the small intestine and MLN were collected for analysis. Percentage of IL-10⁺ cells within a CD45⁺, b CD8⁺, c CD4⁺, d CD19⁺, e IL-7R⁺ Lineage^- (ILCs), f CD11b⁺ and h FOXP3⁺ cells of the MLN and SILP of naïve and D7 infected mice. g Percentage of FOXP3⁺ cells within the CD4⁺ population in the MLN and SILP of naïve and D7 infected mice. Changes in proportion of IL-10 producing cells from total CD45⁺ IL-10⁺ cells in the (i) SILP and (j) MLN from naïve and D7 infected mice. Graphed data are shown with mean ± 1 SD and are pooled from 2–4 independent experiments with n = 2–5 per experiment. Statistical significance was calculated by Student’s t test where data were normally distributed (a, c (SILP)) and Mann–Whitney U test where data were not normally distributed (b, c (MLN), d–f). (Significance *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001).