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. 2022 Oct 26;47(12):3805–3816. doi: 10.1007/s11064-022-03763-1

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — The role of LA in SNI induced glial cell reactions. (A) The GFAP intensity of lumbar 4th /5th dorsal spinal cord in SNI rats was determined by the immunofluorescence with DAPI counterstaining. The scale bar represents 50 μm. (B) LA decreased the SNI induced GFAP-positive cells. (C) The IBA-1 intensity of lumbar 4th / 5th DRGs in SNI rats was determined by the immunofluorescence with DAPI counterstaining. The scale bar represents 100 μm. (D) LA decreased the SNI induced IBA-1-postive cells. n = 4 rats for each group. *P < 0.05 SNI group compared with the control group; and ^#P < 0.05 LA + SNI group compared with SNI group at each day. All data are presented as mean ± SEM. (GFAP for satellite glial cell; IBA-1 for activated macrophage)