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. 2022 Oct 26;47(12):3805–3816. doi: 10.1007/s11064-022-03763-1

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Role of LA on inflammatory cytokines in SN of SNI rats. (A) Western blot analyses showing the LA reversed the changes in the expression of TSLP, TSLPR, IL-33, IL-6 and IL-1β on the 7th day after SNI in SNs of rats. β-actin was used as a loading control. (B-F) showing the relative band density quantified with its own β-actin of indicated band in (A). n = 4 rats for each group. *P < 0.05 SNI group compared with the control group; and ^#P < 0.01 LA + SNI group compared with SNI group at indicated day. All data are presented as mean ± SEM. The tissues were collected on the 7th day after SNI. SN: sciatic nerve