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. 2022 Nov 9;54(11):1901–1912. doi: 10.1038/s12276-022-00886-x

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Heatmap showing the differential expression of the indicated EGFR ligands between THP1-Control (Con) cells and THP1-PM2.5, THP1-PM10, and THP1-KRPM cells identified by RNA-seq. b qRT‒PCR analysis of THP1 cells treated with the indicated concentrations of different types of PM for 24 h. c ELISA of THP1 cells treated with the indicated concentrations of different types of PM for 24 h. AD Arizona dust. d Immunoblot analysis of THP1 cells treated with the indicated concentrations of different types of PM for 24 h. e qRT‒PCR analysis of human cord blood-derived macrophages treated with 5 μg/cm² PM for 24 h. f ELISA of human cord blood-derived macrophages treated with 5 μg/cm² PM for 24 h. *P ≤ 0.05, **P ≤ 0.01, ***P ≤ 0.001.