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. 2022 Aug 9;158(6):545–559. doi: 10.1007/s00418-022-02145-6

Table 3.

Presence of genetically aberrant (aneusomic) cells in the different cellular compartments of premalignant CIN lesions as detected by FISH targeting of SOX2 copy numbers

Histological classification SOX2 pattern Number of areasa Size of aberrant area (Para)basal cell layers Intermediate cell layers Superficial cell layers
Normal 1 29 Absent N N N
LSIL CIN1 1 16 Absent N N N
2 1 Small A/N A/N N
HSIL CIN1-2 1 2 Small A/N N N
CIN2 1 8 Medium A A/N N
2 5 Large A A N
CIN 2–3 2 4 Large A A N
3 4 Large N A/N N
CIN3 2 8 Large A A N
3 15 Large N A A/N

N Normal (Disomy; copy number for SOX2 = 2), A: Aneusomy (copy number for SOX2 > 2), A/N Aneusomy/Normal (Aneusomic cells mixed with disomic cells), LSIL low-grade squamous intraepithelial lesion, HSIL high-grade squamous intraepithelial lesion. For a detailed description of the genetic classification, see the Supplemental Materials and Methods section. Note that Pattern 3 strongly deviated from Pattern 2. In all 19 cases, the SOX2-negative cells in the basal/parabasal cell layers were disomic for the SOX2 gene, while the SOX2-positive intermediate cell layers showed aneuploidy for both targets in nearly all cases

aNot all cases mentioned in Table 2 were available for FISH analysis. Only the number of analyzed areas is indicated.