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. 2022 Nov 2;13(4):649–669. doi: 10.1007/s13167-022-00300-6

Table 4.

Histone methyltransferases and demethylases with reported functions and representative methyltransferase/demethylase inhibitors in lung cancer (including NSCLC)

Enzymes and their inhibitors Target Function Reference
Histone methyltransferases
  MLL2 H3K4 Loss of expression and deleterious mutations in NSCLC [145]
  SMYD2 H3K36 Contributed to NSCLC cell growth [148]
  SETD2 H3K36 Deleterious mutations in primary NSCLC [149]
  DOT1L H3K79 Contributed to NSCLC cell growth [167]
  PRMTs Arginine on H3 and H4 Contributed to NSCLC cell growth and overexpressed in TKI-resistant NSCLC [164]
Histone demethylases
  KDM2A H3K36me2/me1 Overexpressed in NSCLC [157]
  PAD4 Arginine on H3 and H4 Overexpression led to gefitinib resistance in NSCLC [164]
  KDM3 H3K9me2/me1 Overexpressed in NSCLC [159]
EZH2 inhibitors
Clinical Trial Number
  DZNep SAH hydrolase inhibitor (Ki = 50 pM) NA [168]
  GSK2816126 (GSK126) SAM-competitive EZH2 inhibitor (IC50 = 9.9 nM NCT02082977 [169]
  EPZ6438 (Tazemetostat) SAM-competitive EZH2 inhibitor (Ki = 2.5 nM) (IC50 = 11 nM) NCT01897571 NCT02601950 NCT02601937 [170]
  CPI1205 SAM-competitive EZH2 inhibitor NCT02395601 [171]
LSD1 inhibitor
  RG6016 (ORY-1001) FAD-dependent irreversible LSD1 inhibitor (IC50 b 20 nM) NCT02913443 [172]