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. 2022 Nov 23;13:1027756. doi: 10.3389/fimmu.2022.1027756

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Copyright © 2022 Jin, Duan, Du, Xing, Peng and Zhao

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The potential molecular mechanisms of inflammatory effects (right) and immune responses (left) in aneurysm-related subarachnoid hemorrhage. SAH, subarachnoid hemorrhage; DCs, dendritic cells; IL-6, interleukin 6; ROS, reactive oxygen species; MMP, matrix metalloproteinases; NLRP3, NOD-like receptor family pyrin domain-containing 3; TXNIP, thioredoxin-interacting protein; TNF-α, tumor necrosis factor-α; INF-γ, interferon-γ; NKs, natural killer cells; NADPH, nicotinamide adenine dinucleotide phosphate oxidase; Tregs, regulatory T cells.