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. 2022 Dec 13;28:155. doi: 10.1186/s10020-022-00526-0

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — hUCMSCs@Ex-4 promotes the repair of islet tissue damage by reducing the blood glucose level in NOD mice. A Body weight of NOD mice after different treatments. B Blood glucose level in NOD mice with different treatments. C Blood glucose level in NOD mice with different treatments measured by OGTT. D HE staining for islet morphology in NOD mice with different treatments (400 ×). E Immunohistochemical staining analysis of the infiltration of immune cells (CD4⁺ T, CD8⁺ T, CD11b⁺, and CD11c⁺) in the pancreatic tissues of NOD mice with different treatments. F Proportion of β-cells in the pancreatic tissues of NOD mice with different treatments analyzed by immunohistochemical staining. G Proportion of α-cells in the pancreatic tissues of NOD mice with different treatments analyzed by immunohistochemical staining. In panels A-C, *p < 0.05 vs. NOD mice. ^#p < 0.05, NOD mice treated with hUCMSCs@Ex-4 vs. NOD mice treated with hUCMSCs. In panels E–G, *p < 0.05 vs. PBS-treated mice. ^#p < 0.05 vs. NOD mice. n = 12