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. 2022 Dec 2;10:82–86. doi: 10.5414/CNCS110943

Table 2. Histologic findings in published case series.

Markowitz et al. (2002) [3] Li and Verani (2008) [9] Wu et al. (2013) [7] Salvatore et al. (2015) [8]*
Light microscopy
   Global glomerulosclerosis, % 85 24
   Nodular mesangial matrix, % 100 100 100 100
   Thickening of glomerular basement membrane, % 100 100 100
   Glomerulomegaly, % 100 100 100 100
   Interstitial fibrosis and/or tubular atrophy, % 100 100 100 100
      Mild (< 25%) 52 20 20 Mean 48
(35 – 60)
      Moderate (25 – 50%) 61 53 30
      Severe (> 50%) 22 27 50
   Arteriosclerosis, % 100 100 100
      Mild 17 0 0
      Moderate 61 73 25
      Severe 28 27 75
   Hyalinosis and arteriolosclerosis, % 100 100 100
   Mild 4 0 20
   Moderate 57 53 70
   Severe 39 47 10
Immunofluorescence
   Immune deposits / reactivity, % 0 0 0 0
   Non-specific IgM/C3, % 70 80 Infrequent and unspecific patterns, IgG, IgM, C3, and albumin
   Linear staining of GBM and TBM for albumin and IgG, % 44 IgG, 48 albumin 53
Electron microscopy
   Dense deposits, % 0 0 0
   Fibrils, % 0 0
   GBM thickening, % 100 100 100 100
   Foot process effacement, % 46 (fusion) 100 100 100
   Diffuse, % 67 65
   Segmental, % 33 35 (focal)

*Six patients were excluded for the absence of nodular glomerulosclerosis pattern. GBM = glomerular basement membrane; TBM = tubular basement membrane.