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. 2022 Nov 30;9:865788. doi: 10.3389/fmolb.2022.865788

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Copyright © 2022 Li, Han, Zhao, Huang, Feng, Huang, Lan, Huang, Wang, Shen, He, Li, Song, Wang and Meng.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Enrichment analysis of differentially expressed genes (DEGs) in stroke and PSD patients. (A). Biological processes: F2C represents the genes common to stroke and PSD; F2D represents genes that potentially mediate PSD based on excessive changes in expression during stroke recovery; F2E represents depression-specific genes. (B). GSEA. Panels (a,b) use common and specific genes associated with stroke and PSD patients as the preset gene set for GSEA. Panels (c,d) use depression-specific genes as the preset gene set for GSEA. (C). KEGG pathway: F2C represents the genes common to stroke and PSD; F2D represents genes that potentially mediate depression based on excessive changes in expression during stroke recovery; F2E represents depression-specific genes. (D). GSEA-KEGG pathway: the KEGG pathways were divided into four types according to the classification of intestinal flora, signaling pathways, brain disease, and metabolism (from left to right).