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. 2021 Sep 12;38(6):1027–1045. doi: 10.1007/s10565-021-09650-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — CTSLP8 promoted proliferation, glycolysis, and DDP resistance in OC cells. A RT-PCR showed the expression levels of CTSLP8 in four OC cell lines. B RT-PCR verified the effects of lentiviruses transfection with CTSLP8 overexpression (OE) or knockdown (KD). C–D CCK8 showed the effects of CTSLP8-OE (P8-OE) or CTSLP8-KD (P8-KD) on the proliferation of OC cell lines. E–F The clone formation showed the effects of P8-OE or P8-KD on the proliferation of OC cell lines. G ECAR assay showed the effects of P8-OE or P8-KD on the glycolysis of OC cell lines. H The detection of glycolytic products confirmed the effects of P8-OE or P8-KD on the glycolysis of OC cell lines. I–J IC₅₀ assay validated the effect of P8-OE or P8-KD on cisplatin resistance of OC cells