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. 2022 Dec 15;15(1):2155018. doi: 10.1080/19490976.2022.2155018

Figure 6.

Figure 6

:Exposure of PBMCs to patient-derived BEs resulted in altered T cell subtype counts. Th17 (CD3+CD4+IL-17A+), Th1 (CD3+CD4+IFN-γ+), monocytes (CD3−CD19−CD14+) in response to BE exposure from HCs and HBV-CLD patients (a). Th17 (b), Th1 (c) and monocytes (d) in response to BE exposure from HCs, non-cirrhotic (NC) and cirrhotic (Crrh) patients. Data are shown as % of viable CD3+CD4+ lymphocytes for Th17 and Th1 cells, and % of viable singlets for monocytes. Box plots indicate individual values for each sample and median with interquartile range within groups. *p < .05, **p < .01, ***p < .001, ****p < .0001. BE, bacterial extracts; Crrh, cirrhosis; HBV-CLD, hepatitis B virus related chronic liver diseases; HC, healthy control; NC, non-cirrhosis; Th1, T helper 1; Th17, T helper 17.