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. 2022 Dec 17;22:614. doi: 10.1186/s12903-022-02583-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Upregulated VNNs were positively correlated with inflammation. A Correlation between expression levels of VNN1 and IL1B, and between expression levels of VNN2 and IL1B, in gingival tissues of human periodontitis in RT-qPCR (n = 30). B Correlation between VNN1 and IL1B expression levels, and between VNN2 and IL1B expression levels, in periodontitis in GSE10334 (n = 183). C Correlation between VNN1 and IL1B expression levels, and between VNN2 and IL1B expression levels, in periodontitis in GSE16134 (n = 241). Significance was defined in the figure as *** p < 0.001, and **** p < 0.0001