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. 2022 Mar 11;92(6):1555–1565. doi: 10.1038/s41390-022-01955-7

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Representative images of Ki67 immunohistochemical staining in the left ventricle + septum (LV + S; a–c) and right ventricle (RV; d–f) from aged-matched fetal lambs (fetal control, n = 7) and preterm lambs exposed antenatally to saline (preterm SAL, n = 9) or LPS (preterm LPS, n = 9). Scale bar = 100 μm. Graphs show the proportion of Ki67 positively-labeled cardiomyocytes in the LV + S (g) and RV (h) of each experimental group (gray circles = fetal control; blue squares = preterm SAL; orange triangles = preterm LPS). Statistical analyses were performed using a one-way ANOVA followed by a Bonferroni post hoc test. Data are presented as mean ± SD, * denotes p < 0.05, *** denotes p < 0.001.