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. 2022 Dec 22;12:523. doi: 10.1038/s41398-022-02281-6

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — The Cox proportional hazard regression models were applied to estimate the effect of different levels of serum CRP on the incidence of AD among different genotypes of SPI1, CD33, and CLU after adjusting for age, sex, years of education, APOE ε4 and PCs. The results from UKBB are shown (a–c), and those from FHS are shown (d–f). The results from the meta-analyses of UKBB and FHS are shown (g–i). Raw P values are presented. For all CRP cutoffs from 3–12 mg/L, refer to Fig. S2.