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. 2022 Dec 19;14(24):5394. doi: 10.3390/nu14245394

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Tamoxifen, naringenin and quercetin regulated cell cycle progression in HepG2 cells. Cells were exposed to tamoxifen (20 μM), naringenin (10 and 200 μM) and quercetin (10 and 100 μM) either alone or in combination for 24 h. Percentage of cell population in the (A) G0/G1 phase, (B) S phase and (C) G2/M phase, respectively. (D) Tamoxifen, naringenin and quercetin regulated the mRNA transcription of cell cycle-related genes. Results are expressed as mean ± SD of three independent experiments (n = 3). ^a–h: Different letters indicate significant differences (p < 0.05) in cell cycle distribution and their regulated genes.

Tamoxifen, naringenin and quercetin regulated cell cycle progression in HepG2 cells. Cells were exposed to tamoxifen (20 μM), naringenin (10 and 200 μM) and quercetin (10 and 100 μM) either alone or in combination for 24 h. Percentage of cell population in the (A) G0/G1 phase, (B) S phase and (C) G2/M phase, respectively. (D) Tamoxifen, naringenin and quercetin regulated the mRNA transcription of cell cycle-related genes. Results are expressed as mean ± SD of three independent experiments (n = 3). ^a–h: Different letters indicate significant differences (p < 0.05) in cell cycle distribution and their regulated genes.