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. 2022 Dec 23;22:486. doi: 10.1186/s12890-022-02286-3

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — The prognostic value of risk score model. a Principal component analysis depended basically on all genes involved in fatty acid metabolism in LUAD; b Principal component analysis based upon 5 fatty acid metabolism-associated genes from risk score prognostic model; c Kaplan–Meier curves for the overall survival of patients between the higher-risk and lower-risk groups in the TCGA cohort; d Kaplan–Meier curves for the overall survival of patients between the higher-risk and lower-risk groups in the GSE31210 dataset; e Kaplan–Meier curves for the progression free survival of patients between the higher-risk and lower-risk groups in the TCGA cohort; f Kaplan–Meier curves for the progression free survival of patients between the higher-risk and lower-risk groups in the GSE31210 dataset