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. 2022 Dec 24;8:102. doi: 10.1038/s41522-022-00366-0

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — a Schematic of mice experimental design. b, e Mice liver and kidney antioxidant capacity determination, including glutathione peroxidase (GSH-Px) activity, superoxide dismutase (SOD) activity, thioredoxin reductase (TrxR) activity, total antioxidant (T-AOC) capacity, malondialdehyde (MDA) content, total carbonyl (Carbonyl) content. c, f The relative expression of GSH-Px, SOD, Nrf2, TrxR in liver and kidney tissues of oxidatively damaged mice. d Histomorphological analysis of liver kidney and intestine from different mice groups (original magnification, ×400; scale bar = 50 µm). g Mice serum endotoxin determination. Different letters (a, b, c, d) in the figure indicate significant differences between groups (p < 0.05, one-way ANOVA analysis), and the same letters indicate no significant differences. Statistical analysis between groups was expressed as mean ± SD, error bars were standard deviation.