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Schematic diagram of pharmacological mechanisms of XFZYD pretreatment for neurological deficits in tMCAO mice. XFZYD pretreatment increased colocalization of AQP4 with its anchoring proteins, α-dystroglycan, β-dystroglycan, and Agrin, to enhance AQP4 polarization on the glial endfoot and exchange efficiency of CSF and ISF in the GS. Thus, XFZYD pretreatment can prevent the onset of neurodegeneration in IS.
