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. 2022 Dec 13;12:1074538. doi: 10.3389/fonc.2022.1074538

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Copyright © 2022 Dai, Jahanzaib, Liao, Yao, Li, Teng, Chen and Cheng

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Overall survival (OS) in all patients was examined with Kaplan-Meier plots and log-rank tests. (A) Comparison of four groups (KRAS-G12D, KRAS-G12V, KRAS-G12R, KRAS-Q61) with KRAS mutations. (B) Comparison of patients with KRAS wild-type and those with KRAS mutations. Other groups included KRAS-G12D, KRAS-G12V, KRAS-G12R, and KRAS-Q61. (C) Intercomparison between patients with KRAS-G12D mutation and other patients. Other groups included KRAS wild-type, KRAS-G12V, KRAS-G12R, and KRAS-Q61. (D) Examination of mutations at codons 12 and 61. KRAS-G12D, KRAS-G12V, and KRAS-G12R were all part of the codon 12 group of KRAS. The KRAS-Q61 family consisted of four different variants (KRAS-Q61H, KRAS-Q61R, KRAS-Q61K, and KRAS-Q61L). WT, KRAS wild-type; HR, hazard ratio; The numbers in parentheses represent 95% confidence intervals.