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. 2021 Dec 30;13(1):60–76. doi: 10.1080/21505594.2021.2018767

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© 2022 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — A. hydrophila-induced ER-stress induces HKM apoptosis via JNK-Akt signaling pathway. HKM pre-incubated with SP600125, 124,005 or transfected with sc-siRNA or chop-siRNA were infected with A. hydrophila and at 24 h p.i. (a) phosphorylation of JNK, (b) levels of total and phospho-Akt, and (c) HKM apoptosis was studied. Vertical bars represent mean ± SE (n = 3). Asterisk indicates significant difference between indicated groups (*p < 0.05). HKM, uninfected HKM; HKM+B, HKM infected with A. hydrophila; HKM+SP600125 + B, HKM pre-incubated with SP600125 infected with A. hydrophila; HKM+124005 + B, HKM pre-incubated with 124,005 infected with A. hydrophila; HKM+sc-siRNA, HKM transfected with sc-siRNA (scrambled siRNA); HKM+chop-siRNA+B, HKM transfected with chop-siRNA infected with A. hydrophila.