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. 2022 Dec 14;13:1080403. doi: 10.3389/fimmu.2022.1080403

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Copyright © 2022 Wang, Jian, Xue, Meng, Xia, Li, Xu, Dong, Mao and Zhang

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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MYBL1 enhances the malignant biological behaviors of ccRCC cells. (A) The expression level of MYBL1 in renal cancer cells; (B) Knockdown efficiency of MYBL1 in ccRCC cells; (C, D) CCK8 assay between sh-MYBL1 and control cells; (E) Colony formation assay between sh-MYBL1 and control cells; (F) EdU assay between sh-MYBL1 and control cells; (G) Flow cytometry detecting cell apoptosis between sh-MYBL1 and control cells; (H–J) In vivo assay showed that the inhibition of MYBL1 could also hamper tumor growth in mice. *P < 0.05, **P < 0.01, ***P < 0.001.